Even when nutriment is available, it may be poorly engaged in patients with HIV and AIDS. Intestinal malabsorption and nutrient loss is common. While severe diarrhoea and malabsorption may be due to opportunistic infections or intestinal parasites such as cryptosporidiosis, some of the altered absorption appears to be a consequence of HIV infection itself. The virus has been shown to destruction the intestinal villi, and redness can damage gut tissue and reduce absorption.
Frequently small bowel movement time is accelerated, particularly among children with severe diarrhoea. Enzymes in the intestinal mucosa involved with metabolism and absorption can also be less active.
These changes in the gut seem to attack the body’s aptitude to utilise dietary fat and carbohydrates. A number of studies have reported that people with HIV have high levels of foe-cal fat that is distinct to fat intake or, in one study in children, the proximity of any intestinal infection other than HIV itself.
Frequently small bowel movement time is accelerated, particularly among children with severe diarrhoea. Enzymes in the intestinal mucosa involved with metabolism and absorption can also be less active.
These changes in the gut seem to attack the body’s aptitude to utilise dietary fat and carbohydrates. A number of studies have reported that people with HIV have high levels of foe-cal fat that is distinct to fat intake or, in one study in children, the proximity of any intestinal infection other than HIV itself.
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